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Background A left ventricular (LV) thrombus is a rare but known entity, which is commonly associated with dilated cardiomyopathy and acute anterior myocardial infarction ( 1 ). The presence of a mobile LV thrombus, atrial fibrillation and

procedural risks of AF ablation is thromboembolic stroke ( 4 ) and one potential mechanism for this is dislodgement of thrombus present in the atria by catheters and wires used during the procedure. The thromboembolic risk during an AF ablation procedure is

features suggesting hemodynamic compromise ( Fig. 2A and B ). Figure 2 (A) Echo: Subcostal view showing pericardial effusion (arrowed). (B) Apical four chamber (focused view) showing apical trabeculation and apical thrombus (arrowed

A 78-year-old man presented with chest pain, left bundle branch block and raised troponin levels. Echocardiography demonstrated a 63 mm vein graft aneurysm with mural thrombus ( Fig. 1 A, B and C; Videos 1 , 2 , 3 and 4 ), and a dilated

showed a dilated and poor-functioning right ventricle with a snake-like large mobile thrombus extending from the inferior vena cava ( Video 1 ) to the right atrium and prolapsing to the right ventricle ( Video 2 ) across the tricuspid orifice with each

a massive left atrial thrombus (>4 cm in diameter) ( Fig. 1A, B , C , D and Videos 1 , 2 , 3 and 4 ). Intravenous heparin was initiated, and 5 days later, he was taken to the operating room for planned redo mitral valve replacement and left

), severe tricuspid regurgitation (TR), significant tricuspid stenosis (TS), severe aortic regurgitation (AR) and mild pulmonary regurgitation (PR). There was a large ball-shaped left atrium (LA) thrombus (3.65×3.55 cm), which was freely mobile and

Summary Non-atherosclerotic myocardial infarction (MI) is an important but often misdiagnosed cause of acute MI. Furthermore, non-atherosclerotic MI with concomitant acute stroke and pulmonary embolism due to in-transit thrombus across a

more unstable overnight. The following morning, after the echocardiogram demonstrated significant intra-cardiac thrombus burden, it was deemed too high-risk to proceed with thrombolysis. Surgical removal of the thrombi and closure of the patent foramen

echocardiography confirmed the presence of a massive right atrial thrombus, extending from the superior vena cava ( Fig. 3 , panels A, B, and C). The thrombus was highly mobile, hyper segmented and partially protrusive through the tricuspid valve into the right