heart failure, congenital heart disease and cardiac rejection post-transplantation ( 3 ). Tei index has also been shown to have prognostic value in patients with cardiac amyloidosis and dilated cardiomyopathy ( 4 , 5 ). An acute myocardial infarction
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Sadie Bennett, Chun Wai Wong, Timothy Griffiths, Martin Stout, Jamal Nasir Khan, Simon Duckett, Grant Heatlie, and Chun Shing Kwok
Gowsini Joseph, Tomas Zaremba, Martin Berg Johansen, Sarah Ekeloef, Einar Heiberg, Henrik Engblom, Svend Eggert Jensen, and Peter Sogaard
Introduction Clinical outcomes after an acute myocardial infarction (AMI) are determined by the initial morphological and functional alterations resulting from myocardial necrosis ( 1 , 2 , 3 ). Infarct size (IS) is a strong predictor of
Sergio Barros-Gomes, Abdallah El Sabbagh, Mackram F Eleid, and Sunil V Mankad
Summary Non-atherosclerotic myocardial infarction (MI) is an important but often misdiagnosed cause of acute MI. Furthermore, non-atherosclerotic MI with concomitant acute stroke and pulmonary embolism due to in-transit thrombus across a
Ying X Gue, Sanjay S Bhandari, and Damian J Kelly
Background ST-segment elevation myocardial infarction (STEMI) commonly occurs when a coronary artery becomes totally occluded by a blood clot disrupting blood flow to the myocardium. The UK national registry has shown that out of 80 724
Rienzi Díaz-Navarro and Petros Nihoyannopoulos
Background A pseudoaneurysm (Ps) of the left ventricle (LV) is a severe complication of acute myocardial infarction (AMI) resulting from a free cardiac wall rupture that is contained by the pericardium, thrombus or adhesions ( 1 ). Most
Sathish Kumar Parasuraman PhD MRCP, Janaki Srinivasan, and Paul Broadhurst
is responsible for 15% of all deaths ( 1 , 2 ). In total, 90% of myocardial ruptures happen during the first 2 weeks after acute myocardial infarction ( 3 ). The incidence has gradually fallen due to introduction of reperfusion strategies
Baskar Sekar, Joe Martins, and Sanjiv Petkar
pain to be due to indigestion and kept taking antacids without much symptomatic relief. Clinical examination on arrival was unremarkable. Admission 12 lead electrocardiogram (ECG) was diagnostic of a recent anterolateral myocardial infarction (MI) ( Fig
Thomas Sturmberger, Johannes Niel, Josef Aichinger, and Christian Ebner
systolic longitudinal strain of the lateral and posterior walls ( Fig. 2 ; bull’s eye). Elevated cardiac enzymes and decreased longitudinal systolic shortening usually favor the diagnosis of acute coronary syndrome, but in this young patient, myocardial
Jonathan Hinton, George Hunter, Madhava Dissanayake, and Rob Hatrick
Learning points: Pseudo-aneurysms are a rare but life-threatening complication of a myocardial infarction and may present at any time point following a myocardial infarction. Patients with a chronic pseudo-aneurysm will often remain
Lijun Qian, Feng Xie, Di Xu, and Thomas R Porter
events (MACE), including the separate detection of nonfatal myocardial infarction (NFMI) and death. Data collection Two investigators performed eligibility assessment with a standardized data extraction individually and another reviewer checked