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Theodoros Ntoskas, Farhanda Ahmad, and Paul Woodmansey

for determining the extent of hibernating myocardium ( 2 ). The stressor used for SE can be physical exercise, pacemaker stress, pharmacological agent or a combination of pharmacological agents ( 3 ). Dobutamine is the preferred pharmacological

Open access

Thomas R Porter

Dobutamine stress echocardiography (DSE) has been utilized extensively in the detection of coronary artery disease (CAD) and prediction of patient outcome ( 1 , 2 , 3 , 4 ). Its safety has also been thoroughly investigated in the contemporary

Open access

Patrick H Gibson, Fernando Riesgo, Jonathan B Choy, Daniel H Kim, and Harald Becher

Introduction Dobutamine stress echocardiography (DSE) is commonly performed during follow-up after cardiac transplantation as part of surveillance for the diagnosis of cardiac allograft vasculopathy (CAV). Previous studies have demonstrated the

Open access

Jamal N Khan, Timothy Griffiths, Tamseel Fatima, Leah Michael, Andreea Mihai, Zeeshan Mustafa, Kully Sandhu, Robert Butler, Simon Duckett, and Grant Heatlie

Introduction The expanding responsibilities and skillset of the Highly Specialised Cardiac Echocardiography Physiologist include performance and analysis of exercise and dobutamine stress echocardiography studies ( 1 , 2 ). Physiologist

Open access

Keith Pearce and John Chambers

cardiac physiologist’. He reminds us ‘that the biggest danger associated with dobutamine stress echocardiography is in misinterpreting the data obtained…’. Did Ntoskas et al . ( 2 ) really misinterpret the data? Dr Porter states that 7 patients with

Open access

Liam Ring, Benoy N Shah, Sanjeev Bhattacharyya, Allan Harkness, Mark Belham, David Oxborough, Keith Pearce, Bushra S Rana, Daniel X Augustine, Shaun Robinson, and Christophe Tribouilloy

Fig. 16. Figure 16 Summary of recommendations for DSE in low-gradient AS with impaired LVEF. After exclusion of technical error, such patients should be considered for dobutamine stress echocardiography (DSE). Conventional

Open access

Peter H Waddingham, Sanjeev Bhattacharyya, Jet Van Zalen, and Guy Lloyd

either through physiological means with exercise or pharmacological means such as the beta-adrenergic effects of dobutamine. Dipyridamole is predominantly vasodilatory causing increased coronary flow through inhibition of cellular re-uptake of adenosine

Open access

Benoy N Shah, Anita MacNab, Jane Lynch, Reinette Hampson, Roxy Senior, and Richard P Steeds

( 1 ). As 2-dimensional echocardiography became more widely available, a wealth of data accrued during the 1980s and 1990s demonstrating the feasibility, safety and accuracy of exercise ( 2 ), dobutamine ( 3 ) and dipyridamole ( 4 ) stress

Open access

Benoy N Shah and Roxy Senior

’, ‘significant obesity’ and ‘obesity’ combined with the following: ‘dobutamine echocardiography’, ‘dobutamine stress echocardiography’ (DSE), ‘exercise echocardiography’, ‘stress echocardiography’ (SE), ‘thallium scintigraphy’, ‘technetium scintigraphy’, ‘single

Open access

Adrian Chenzbraun

assessed with a low-dose (5–20 μg/kg per min) dobutamine protocol recording changes in stroke volume (SV), ejection fraction and gradients as appropriate. The dose dependency of the inotropic response is unpredictable and does not necessarily parallel the