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Benoy N Shah and Roxy Senior

necessary to exclude coronary artery disease (CAD) as a possible underlying cause (e.g. angina equivalent as a cause of dyspnea). The body habitus of such patients often poses significant technical challenges for each of the noninvasive imaging techniques

Open access

Maria Pia Donataccio, Claudio Reverberi, and Nicola Gaibazzi

Background The combination of myocardial perfusion (MP) imaging and dipyridamole or dobutamine real-time contrast echocardiography improves the sensitivity to detect coronary artery disease (CAD), particularly multivessel CAD. Patients with diffused

Open access

Alexandros Papachristidis, Damian Roper, Daniela Cassar Demarco, Ioannis Tsironis, Michael Papitsas, Jonathan Byrne, Khaled Alfakih, and Mark J Monaghan

coronary artery disease (CAD) ( 2 ), valvular heart disease ( 3 ), pre-operative assessment ( 4 ) and in the assessment of myocardial viability ( 5 ). The guidelines have recommended the use of imaging functional tests in the diagnosis of intermediate

Open access

P A Patel, K A Ravi, D P Ripley, J Kane, E Wass, A Carr, D Wilson, N Watchorn, R K Hobman, D Gill, W P Brooksby, N Kilcullen, and N Artis

as ‘atypical’ and one or none as ‘non-anginal.’ In those instances where angina cannot be diagnosed or excluded on clinical grounds, a framework is provided to estimate the likelihood of underlying significant coronary artery disease (CAD). This is

Open access

M Alsharqi, W J Woodward, J A Mumith, D C Markham, R Upton, and P Leeson

diagnosis and care of heart failure either related to coronary artery disease or other cardiac pathology. Identification and assessment of systolic heart failure relies on identification of wall motion abnormalities. Quantitative assessment of changes in

Open access

Vasiliki Tsampasian, Vasileios Panoulas, Richard J Jabbour, Neil Ruparelia, Iqbal S Malik, Nearchos Hadjiloizou, Angela Frame MSc, Sayan Sen, Nilesh Sutaria, Ghada W Mikhail, and Petros Nihoyannopoulos

patients with (1) coronary artery disease; (2) atrial fibrillation; (3) severe coexistent valve lesions, including severe aortic regurgitation (AR), mitral regurgitation (MR), mitral stenosis (MS); (4) valve-in-valve procedures (patients that had undergone

Open access

Thomas Sturmberger, Johannes Niel, Josef Aichinger, and Christian Ebner

clinical presentation, ECG changes, elevated cardiac enzymes, and lack of coronary artery disease. Focal myocarditis can mimic acute coronary syndromes, and therefore, invasive coronary angiography is frequently performed in this clinical setting. The

Open access

Thomas Mathew, Lynne Williams, Govardhan Navaratnam, Bushra Rana, Richard Wheeler, Katherine Collins, Allan Harkness, Richard Jones, Dan Knight, Kevin O'Gallagher, David Oxborough, Liam Ring, Julie Sandoval, Martin Stout, Vishal Sharma, Richard P Steeds, and on behalf of the British Society of Echocardiography Education Committee

causes (hypertension, coronary artery disease, excess alcohol consumption, tachycardia-induced cardiomyopathy, systemic or pericardial disease, cor pulmonale and congenital heart disease) have been excluded ( 18 , 19 , 20 ). The value of 117% is

Open access

A J Fletcher, S Robinson, and B S Rana

.01 mmHg. LofA 3.5 to −3.5 mmHg RAP = (1.66 × E/e′) + 2.96 Sade et al. 2007 101 from 89 patients On Cardio-thoracic intensive care unit. 55% had coronary artery disease All Simultaneous Not reported RAP = (1.62 × E/e′) + 2

Open access

Camelia Demetrescu, Shelley Rahman Haley, and Aigul Baltabaeva

with HCM may also have atherosclerotic coronary artery disease (CAD). Reports on the prevalence of CAD in HCM have varied, but up to 20% of adult HCM patients over the age of 45 years have been shown to have coexistent CAD ( 7 ). Severe epicardial CAD